By Carmen Adkins
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Cochrane Database Syst Rev, 8, Cd006826. , Sohal, S. , & Walters, E. H. (2014). Epithelial-mesenchymal transition as a fundamental underlying pathogenic process in COPD airways: fibrosis, remodeling and cancer. Expert Rev Respir Med, 1-13. , Chung K. F. (2008). Fluticasone, but not salmeterol, reduces cigarette smoke-induced production of interleukin-8 in human airway smooth muscle. Pulm Pharmacol Ther, 21(2), 292-297. 001 34 M. Suji Eapen, S. Dhar Shukla, M. Quasir Mahmood et al. Osman, I. , Godden, D.
Indeed, the relationship between COPD pathology and carcinogenesis may reflect a more general paradigm of epithelial instability and cancer aetiology, bearing in mind that epithelial cancers make up 90% of all malignancies (Garber, 2008). , 2008). , 1997; Yao, Wang, Lemon, Lubet, & You, 2004). , 2007). Similar findings were reported in ex-smokers with COPD (Kiri, Fabbri, Davis, & Soriano, 2009). Lee et al. reported ICS are associated with a reduced risk of lung cancer but not of laryngeal cancer (C.
2008). Role of HDAC2 in the Pathophysiology of COPD. Annu Rev Physiol. 163257 Barnes, P. J. (2009). Histone deacetylase-2 and airway disease. Ther Adv Respir Dis, 3(5), 235-243. 1177/ 1753465809348648 26 M. Suji Eapen, S. Dhar Shukla, M. Quasir Mahmood et al. Barnes, P. , & Adcock, I. M. (2011). Chronic obstructive pulmonary disease and lung cancer: a lethal association. Am J Respir Crit Care Med, 184(8), 866-867. 201108-1436ED Barnes, P. , Adcock, I. , & Ito, K. (2005). Histone acetylation and deacetylation: importance in inflammatory lung diseases.
Corticosteroids and Steroid Therapy: New Research by Carmen Adkins