Download Coffee is Good for You: From Vitamin C and Organic Foods to by Robert J. Davis PDF

By Robert J. Davis

ISBN-10: 1101553995

ISBN-13: 9781101553992

Although nutrients is meant to be one in every of life's uncomplicated pleasures, few issues reason extra angst and confusion. on a daily basis we're bombarded with come-ons for the newest vitamin, provides for "clinically proven" miracle materials, and warnings approximately contaminants in our favourite meals. It's sufficient to provide anyone indigestion.

Packed with useful-and surprising-information, Coffee is nice for You cuts throughout the litter to bare what's plausible and what's no longer in a enjoyable and simply digestible way.

You'll locate out:

• in the neighborhood grown produce isn't unavoidably extra healthy than fruit and veggies from around the globe
• Alcohol does reason breast melanoma
• You don't desire 8 glasses of water an afternoon for stable health and wellbeing
• Milk isn't useful for powerful bones
• Oatmeal rather can reduce ldl cholesterol
• Sea salt isn't extra healthy than normal salt
• Low-fat cookies can be worse for you than high-fat cheese

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Read Online or Download Coffee is Good for You: From Vitamin C and Organic Foods to Low-Carb and Detox Diets, the Truth about Diet and Nutrition Claims PDF

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Extra info for Coffee is Good for You: From Vitamin C and Organic Foods to Low-Carb and Detox Diets, the Truth about Diet and Nutrition Claims

Example text

Our hypothesis are that (i) the selected dietary flavonoids will influence the disposition and toxicity of irinotecan via induction of the glucuronidation (by UGT1A1) of its active metabolite, SN-38; and (ii) induction of UGT1A1 by dietary flavonoids is influenced by genetic differences in the promoter region of the UGT1A1 gene. The specific aims are to (1) investigate the in vivo Studies 37 interaction of soy isoflavones, chrysin and quercetin with irinotecan in rats, (2) determine whether hepatic UGT1A1 induction by flavonoids is responsible for their interaction with irinotecan, and (3) investigate the influence of the TATA polymorphism in the promoter region of UGT1A1 on inducibility by these flavonoids.

To do so, TRAMP mice, before developing tumors, and mice bearing different stages of prostate cancer, will be treated with genistein for different time periods. Prostatic histopathology, cell proliferation and cell death will be examined in untreated vs. genistein-treated groups. Second, we will examine possible mechanisms by which genistein inhibits cell proliferation of prostate cancer in TRAMP mice in vivo, and in TRAMP cell lines (C1 and C2) in vitro. Specifically, we will investigate the effects of genistein on growth factor (TGF-alpha and/or IGF-I)-induced expression of cell cycle regulatory proteins and MAPK and PI3K pathways in TRAMP cell lines and in TRAMP dorsolateral prostate.

Recent findings in our laboratories and by our collaborators indicate potential for therapeutic benefit in normal and neoplastic prostate. The goal of this translational application is to determine whether a dietary supplement consisting of whole soy protein can inhibit the proliferation of prostate cancer cells in human patients. This question will be tested in a clinical therapeutic intervention in men with prostate cancer. We hypothesize that dietary soy consumption will decrease proliferation in neoplastic and adjacent normal prostatic tissues, as reflected in elevated expression of cell cycle inhibitory proteins (p27 and p21), increased apoptosis, and decreased expression of proliferation markers (KI67- Studies 29 MIB and cyclin B1).

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Coffee is Good for You: From Vitamin C and Organic Foods to Low-Carb and Detox Diets, the Truth about Diet and Nutrition Claims by Robert J. Davis


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