By C. Gopalan (auth.), Prof. H. K. A. Visser, Dr. J. G. Bindels (eds.)
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Additional resources for Child Nutrition in South East Asia: Yogyakarta, 4–6 April 1989
4. Darwin Karyadi: Establishment of a regional network to strengthen national vitamin A deficiency control programme. Mission findings and recommendation, FAO, STC Report, 1988. 5. Helen Keller International: The effect of diarrheal disease on the absorption and utilization of vitamin A. Information Paper 2 by Donna Nager, 1979. 6. Sommer A: The Lancet 1:1411,1980. 7. : Am J Clin Nutr 40: 1090, 1984. 8. Nauss KM: In: Bauernfeind JC (ed) Vitamin A deficiency and its control, Academic Press Inc, Orlando, 1986, p.
The enhanced clinical responses are attributed to alterations in the host-specific and non-specific immune competence. On the other hand, infectious organisms are capable of altering the nutritional state of animals inoculated by natural or unnatural routes. Animal studies are fundamental for understanding infection-malnutrition interactions, but they may not be extrapolated to human situations because (a) experimental nutritional deficiencies leading to host resistance in animals do not easily occur in human populations; and (b) the type, dose, specificity, and route of infection used in animal models may not be the same when observed naturally in humans.
In the pancreas, there is a release of insulin and glucagon and enhanced glucose utilization for maintenance of fever; this, in tum, diminishes utilization of free fatty acids and ketones for energy sources. Clinical findings of persons suffering from infections relate to the actions unleashed by interleukin-l, for instance, fever, anorexia and malaise, the first responses to infection. There is an acceleration of metabolism of body cells, resulting in marked loss of muscle, fatigue and weakness; there is leukocytosis.
Child Nutrition in South East Asia: Yogyakarta, 4–6 April 1989 by C. Gopalan (auth.), Prof. H. K. A. Visser, Dr. J. G. Bindels (eds.)